Ruslan Soldatov @_ruslansoldatov
Computational biologist, Assistant member @MSKCancerCenter componcmsk.org/quantitative-c… NYC Joined June 2021-
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Ruslan Soldatov retweeted
Our paper is now out in final form at Nature Genetics! For those who missed the preprint, we used large-scale Perturb-seq targeting transcription factors to push primary fibroblasts into diverse transcriptional states, including those observed in cell atlas studies.
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Ruslan Soldatov retweeted
Finally out in Nature our collaboration with the Velten Lab 🥰 Enjoy the reading! nature.com/articles/s4158…
Ruslan Soldatov retweeted
What protects individuals from developing blood cancers? Thrilled to share my work in @bloodgenes lab, describing inherited resilience protecting blood stem cells from clonal hematopoiesis by modifying RNA regulation. 🧵👇(1/n) biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
I’m excited to share my work over the last few years with @JD_Buenrostro! Chronic inflammation creates an epigenetic memory in colonic stem cells, priming them for tumor growth: tinyurl.com/ColitisNagaraj…. Here’s a walkthrough of what we found (🧵)
Ruslan Soldatov retweeted
PERFF-seq is out today in @NatureGenet! Our recent thread summarizes why we are excited about the prospects of programmable nucleic acid cytometry for studying rare cell states. nature.com/articles/s4158…
PERFF-seq is out today in @NatureGenet! Our recent thread summarizes why we are excited about the prospects of programmable nucleic acid cytometry for studying rare cell states. nature.com/articles/s4158…
Ruslan Soldatov retweeted
Several years in the making (and aging), @Xueqian_Zhuang 's study on aging is out today @Nature. She finds, surprisingly, that aging suppresses tumorigenesis and stemness in the lung by promoting a functional iron insufficiency in the alveolar epithelium. nature.com/articles/s4158…
Ruslan Soldatov retweeted
(1) How can we build complex structures from a limited set of progenitors and cell types? In our new preprint, we describe patterns of cell fate decisions during early development and (in some cases) identify molecular correlates of different behaviors. biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
🎉Excited to present our latest work out today @Nature 1.What gives a leukemia its phenotype – the oncogenic driver or the differentiation stage of the cell-of-origin? 2.Why do RAS mutations always happen late in AML? 3.Who will relapse after VEN? nature.com/articles/s4158…
Ruslan Soldatov retweeted
Thrilled to share our paper now out @Nature delineating #plasticity during #CRC #metastasis, a fantastic collaboration led by @andrewrmoorman @elliebenitez11, and @f_cambuli from the @KarunaGaneshLab and @Dana_Per lab! nature.com/articles/s4158… rdcu.be/dYAVK. A 🧵
Ruslan Soldatov retweeted
Out today, our exciting study showing how TGFb secretion by Tet2-mutant CH macrophages is a druggable resistance mechanism in Anaplastic Thyroid Cancer. @rosslevinemd @sanchezvelamd @RichardKoche @MSKCancerCenter
Out today, our exciting study showing how TGFb secretion by Tet2-mutant CH macrophages is a druggable resistance mechanism in Anaplastic Thyroid Cancer. @rosslevinemd @sanchezvelamd @RichardKoche @MSKCancerCenter
Ruslan Soldatov retweeted
Our latest preprint is out! 🚨We discovered that intestinal stem cells actively fight bacterial infections. Learn more about it in our new study by @SachaLEBON in collaboration with @matanhofree biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
new out in Nature nature.com/articles/s4158…
Ruslan Soldatov retweeted
Can we recreate the transcriptional states seen in single-cell atlas projects? In our new preprint we try to do this using large-scale Perturb-seq experiments. Along the way we discuss off-target effects of CRISPR epigenetic editors and identify drivers of key fibroblast states.
Ruslan Soldatov retweeted
Excited to share our preprint "Multiome Perturb-seq unlocks scalable discovery of integrated perturbation effects on the transcriptome & epigenome". This work extends single-cell CRISPR screens to simultaneously profile gene expression & chromatin accessibility.
Ruslan Soldatov retweeted
On bioRxiv today, we raise serious concerns over a recent @Nature paper describing ReDeeM, a new single-cell mitochondrial DNA (mtDNA) sequencing / analysis method. Here are three things that you need to know: 1/n biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
Clonal expansions can remodel entire tissues even when they appear normal, but what mechanisms drive this? And how do these processes differ from tumorigenesis? Out @Nature, we identify distinct TNF programs during tumor evolution. 🧵 nature.com/articles/s4158…
Ruslan Soldatov retweeted
Excited to share our newest pre-print by Xueqian Zhuang on how aging suppresses tumorigenesis and stemness in the lung by promoting a functional iron insufficiency in the alveolar epithelium. Interestingly, this renders aged cells resistant to ferroptosis: biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
Tomorrow, 2024-06-24, at 18:00 CEST will be another community meeting! @LouisFaure_ will talk about about scFates (github.com/LouisFaure/scF…), a scalable Python suite for fast tree inference and advanced pseudotime downstream analysis, with a focus on fate biasing.
Ruslan Soldatov retweeted
Excited to share our lab’s latest preprint exploring how mutations and transcriptional cell states cooperate to drive therapeutic resistance in Ph+ B cell acute lymphoblastic leukemia (Ph+ ALL)! (1/14) biorxiv.org/content/10.110…
Ruslan Soldatov retweeted
Excited to share our latest paper led by @rachelaglenn. We generated a genetically diverse mouse pluripotent stem cell panel from the Diversity Outbred stock and outline experimental approaches for "population-scale" in vitro directed differentiation. biorxiv.org/content/10.110…
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